Progression of diabetic nephropathy Role of plasma homocysteine and plasminogen activator inhibitor-1

article
Among patients with diabetic nephropathy, the decline in glomerular filtration rate (GFR) varies substantially, ranging from 2 to 20 mL/min per year. Identification of predictors of progression in diabetic nephropathy is important. Plasma total homocysteine (tHcy) rises with urinary albumin excretion rate in diabetes, and plasminogen activator inhibitor-1 (PAI-1) has been correlated with increased matrix accumulation in various glomerulopathies. In this prospective observational cohort study, we evaluated the importance of baseline tHcy and PAI-1 as predictors of the rate of decline in GFR. Baseline tHcy and PAI-1 were measured in 157 type 1 diabetic patients with diabetic nephropathy (92 men; mean age, 41 ±: 10 years; mean diabetes duration, 27 ± 8 years; median GFR, 80 mL/min/1.73 m2 [range, 23 to 143 mL/min/1.73 m2]). Hereafter, GFR was measured yearly with a plasma clearance technique for at least 3 years (median, 7 years [range, 3.0 to 8.3 years]). The mean rate of decline in GFR was 3.7 ± 0.3 mL/min per year. A linear regression analysis revealed a borderline significant relationship between rate of decline in GFR and tHcy (P = 0.069) and PAI-1 (P = 0.087). Analysis of the rate of decline in GFR and tertiles of tHcy and PAI-1 revealed that increasing levels of tHcy were correlated with a significantly faster decline in GFR (P = 0.025), whereas increasing levels of PAI-1 were not. After adjustment for other well-established risk factors for progression of nephropathy in a multiple linear regression analysis, however, neither tHcy levels nor PAI-1 levels were independent predictors of rate of decline in GFR. © 2001 by the National Kidney Foundation, Inc. Chemicals / CAS hemoglobin A1c, 62572-11-6; homocysteine, 454-28-4, 6027-13-0; plasminogen activator inhibitor 1, 140208-23-7
TNO Identifier
953570
ISSN
02726386
Source
American Journal of Kidney Diseases, 38(6), pp. 1376-1380.
Pages
1376-1380
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