Specific and efficient targeting of adenovirus vectors to macrophages: Application of a fusion protein between an adenovirus-binding fragment and avidin, linked to a biotinylated oligonucleotide

article
Background: The application of serotype 5 adenoviruses (Ad5) in macrophages is hampered by the absence of the endogenous coxsackie adenovirus receptor (CAR). Methods: To overcome this limitation, we first generated a linker protein consisting of the virus-binding-domain of CAR and the C-terminus of avidin. Second, to target macrophages, this linker protein was equipped with the biotinylated (bio) oligonucleotide dA6G10, which was previously shown to display a high affinity for the scavenger receptor A (SR-A). Results: As compared to nontargeted virus, the linke r protein equipped with bio-dA6G10 showed a 500-fold increased reporter gene expression in mouse macrophage RAW264.7 cells. A linker protein equipped with a bio-dA16 control oligonucleotide was inactive. Moreover, the bio-dA6G10-equipped linker showed a 390-fold increased luciferase expression in the macrophage cell line J774 and 276- and 150-fold increased reporter gene expression in primary peritoneal and bone marrow (BM)-derived macrophages, respectively. Using BM-derived macrophages from SR-A knockout mice, it was shown that the dA6G10-mediated uptake is predominantly SR-A-mediated. Conclusions: Thus, we have developed a novel tool to link biotinylated ligands to a virus-binding fragment of CAR and have exploited this linker protein to extend the applicability of Ad5 to infect transformed and primary macrophages. Copyright © 2006 John Wiley & Sons, Ltd. Chemicals / CAS: Avidin, 1405-69-2; CAR receptor; Ligands; Oligonucleotides; Receptors, Virus; Recombinant Fusion Proteins
TNO Identifier
239292
ISSN
1099498X
Source
Journal of Gene Medicine, 8(6), pp. 668-678.
Pages
668-678
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