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Differential effects of amlodipine and atorvastatin treatment and their combination on atherosclerosis in ApoE*3-Leiden transgenic mice
Differential effects of amlodipine and atorvastatin treatment and their combination on atherosclerosis in ApoE*3-Leiden transgenic mice
Rosuvastatin reduces atherosclerosis development beyond and independent of its plasma cholesterol-lowering effect in APOE*3-Leiden transgenic mice: Evidence for antiinflammatory effects of rosuvastatin
Rosuvastatin reduces atherosclerosis development beyond and independent of its plasma cholesterol-lowering effect in APOE*3-Leiden transgenic mice: Evidence for antiinflammatory effects of rosuvastatin
CD36 deficiency increases insulin sensitivity in muscle, but induces insulin resistance in the liver in mice
CD36 deficiency increases insulin sensitivity in muscle, but induces insulin resistance in the liver in mice
Clearance of coagulation factor VIII in very low-density lipoprotein receptor knockout mice
Clearance of coagulation factor VIII in very low-density lipoprotein receptor knockout mice
Hepatic Steatosis: A Mediator of the Metabolic Syndrome. Lessons from Animal Models
Hepatic Steatosis: A Mediator of the Metabolic Syndrome. Lessons from Animal Models
Overexpression of APOC1 in obob mice leads to hepatic steatosis and severe hepatic insulin resistance
Overexpression of APOC1 in obob mice leads to hepatic steatosis and severe hepatic insulin resistance
ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglycerides (VLDL-TG) production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis
ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglycerides (VLDL-TG) production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis
The VLDL receptor plays a major role in chylomicron metabolism by enhancing LPL-mediated triglyceride hydrolysis
The VLDL receptor plays a major role in chylomicron metabolism by enhancing LPL-mediated triglyceride hydrolysis
Apolipoprotein AV: Low concentration, high impact
Apolipoprotein AV: Low concentration, high impact
Response of apolipoprotein E*3-Leiden transgenic mice to dietary fatty acids: Combining liver proteomics with physiological data
Response of apolipoprotein E*3-Leiden transgenic mice to dietary fatty acids: Combining liver proteomics with physiological data
Rosuvastatin reduces plasma lipids by inhibiting VLDL production and enhancing hepatobiliary lipid excretion in ApoE*3-Leiden mice
Rosuvastatin reduces plasma lipids by inhibiting VLDL production and enhancing hepatobiliary lipid excretion in ApoE*3-Leiden mice
No effect of C-reactive protein on early atherosclerosis development in apolipoprotein E*3-Leiden/human C-reactive protein transgenic mice
No effect of C-reactive protein on early atherosclerosis development in apolipoprotein E*3-Leiden/human C-reactive protein transgenic mice
Intracerebroventricular administration of melanotan II increases insulin sensitivity of glucose disposal in mice
Intracerebroventricular administration of melanotan II increases insulin sensitivity of glucose disposal in mice
Apolipoproteins modulate the inflammatory response to lipopolysaccharide
Apolipoproteins modulate the inflammatory response to lipopolysaccharide
Efficient targeting of adenoviral vectors to integrin positive vascular cells utilizing a CAR-cyclic RGD linker protein
Efficient targeting of adenoviral vectors to integrin positive vascular cells utilizing a CAR-cyclic RGD linker protein
Tumor necrosis factor-α plays an important role in restenosis development
Tumor necrosis factor-α plays an important role in restenosis development
Apolipoprotein CI overexpression is not a relevant strategy to block cholesteryl ester transfer protein (CETP) activity in CETP transgenic mice
Apolipoprotein CI overexpression is not a relevant strategy to block cholesteryl ester transfer protein (CETP) activity in CETP transgenic mice
Local perivascular delivery of anti-restenotic agents from a drug-eluting poly(ε-caprolactone) stent cuff
Local perivascular delivery of anti-restenotic agents from a drug-eluting poly(ε-caprolactone) stent cuff
ApoC-III deficiency prevents hyperlipidemia induced by apoE overexpression
ApoC-III deficiency prevents hyperlipidemia induced by apoE overexpression
Triglyceride-rich lipoprotein metabolism in unique VLDL receptor, LDL receptor, and LRP triple-deficient mice
Triglyceride-rich lipoprotein metabolism in unique VLDL receptor, LDL receptor, and LRP triple-deficient mice
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